Journal of Brain and Neurology

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Rapid Communication - Journal of Brain and Neurology (2023) Volume 6, Issue 1

multiple sclerosis in the of antibody-associated demyelinating syndromes in brain

Kévin Bigaut*

Department of Clinical Pharmacy, University of Medical Sciences, Iraq

*Corresponding Author:
Kévin Bigaut
Department of Clinical Pharmacy
University of Medical Sciences, Iraq

Received: 30-Dec-2022, Manuscript No. AAJBN-23-86605; Editor assigned: 03-Jan-2023, PreQC No. AAJBN-23-86605(PQ); Reviewed: 17-Jan-2023, QC No. AAJBN-23-86605; Revised:24-Jan-2023, Manuscript No. AAJBN-23-86605(R); Published:31-Jan-2023, DOI:10.35841/aajbn-6.1.135

Citation: Bigaut K. Multiple sclerosis in the of antibody-associated demyelinating syndromes in brain. J Brain Neurol. 2023;6(1):135

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Multiple Sclerosis (MS) is a potentially disabling disease of the brain and spinal cord.In MS, the immune system attacks the protective sheath that covers nerve fibres and causes communication problems between your brain and the rest of your body. Eventually, the disease can cause permanent damage or deterioration of the nerves vary widely and depend on the amount of nerve damage and which nerves are affected. Some people with severe MS may lose the ability to walk independently or at all, while others may experience long periods of remission without any new symptoms. There's no cure for multiple sclerosis. However, treatments can help speed recovery from attacks, modify the course of the disease and manage symptoms. Multiple sclerosis signs and symptoms may differ greatly from person to person and over the course of the disease depending on the location of affected nerve fibres. Numbness or weakness in one or more limbs that typically occurs on one side of your body at a time or your legs and trunk electric-shock sensations that occur with certain neck movements, especially bending the neck forward lack of coordination or unsteady gait. Genetic factor includes Susceptibility may pass down in the genes, but scientists believe an environmental trigger is also necessary for MS to develop, even in people with specific genetic features [1].

People who smoke appear to be more likely to develop they also tend to have more lesions and brain shrinkage than non-smokers. Infections are Exposure to viruses, such as Epstein-Barr Virus (EBV) or mononucleosis, may increase trusted Source a person’s risk of developing but research has not shown a definite link. Other viruses that may play a role include human herpes virus type 6 (HHV6) and mycoplasma pneumonia. Vitamin D deficiency is more common among people who have less exposure to bright sunlight, which is necessary for the body to create vitamin D. Some experts think that low levels of vitamin D may affect the way the immune system works. Vitamin B12 deficiency in The body uses vitamin B when it produces myelin. A lack of this vitamin may increase Trusted Source the risk of neurological diseases such as previous theories have included exposure to canine distemper, physical trauma, or aspartame, an artificial sweetener, but there is no evidence to support .There is probably no single trigger for MS, but multiple factors may contribute [2].

Diagnosis will carry out a physical and neurological examination ask about symptoms and consider the person’s medical history. No single test can confirm a diagnosis, so a doctor will use several strategies when deciding whether a person meets the criteria for a diagnosis. MRI scans of the brain and spinal cord which may reveal lesions. Spinal fluid analysis, which may identify antibodies that suggest a previous infection or proteins consistent with a diagnosis of MS an evoked potential test which measures electrical activity in response to stimuli. Other conditions have symptoms that are similar to those of MS, so a doctor may suggest other tests to assess for other possible causes of the person’s symptoms. If the doctor diagnoses MS, they will need to identify what type it is and whether it is active or not. The person may need more tests in the future to check for further changes [3].

Multiple Sclerosis (MS) is a constant safe intervened demyelinating confusion of the focal sensory system. It typically presents in early adulthood however in of cases side effects start before the addressing paediatric MS Beginning before adolescence is considerably more uncommon just of cases start before. After its most memorable portrayal by Charcot in practically all repetitive CNS demyelinating disorders had been characterized under the umbrella finding of up to this point para clinical markers opened up. Anyway the enormous fluctuation in clinical show reaction to treatment and result of MS, especially among paediatric patients, is deep rooted. The depiction of hostile to aquaporin and against myelin oligodendrocyte glycoprotein upholds the chance of misdiagnosed cases inside past series. The conclusion is significantly seriously testing in exceptionally youthful patients, for whom hereditary and metabolic examinations are frequently expected to avoid specific MS imitates. Advances and accessibility of demonstrative strategies somewhat recently legitimize the assessment of late paediatric cases. We inspected the attributes of paediatric MS determined after to have the cooperation of essentially all paediatric nervous system science focuses across and contrasted them and our recently distributed paediatric accomplice analyzed before We additionally inspected information for contrasts between youngsters old and those matured years at beginning of side effects [4,5].


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