The Cognitive Neuroscience Journal

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Rapid Communication - The Cognitive Neuroscience Journal (2022) Volume 5, Issue 3

Role of combined cognitive bias hypothesis in anxiety

Joao Setubal*

Department of Behavior and Cognitive Neuroscience, School of Psychology, Shaanxi Normal University, Xi’an, China

*Corresponding Author:
Joao Setubal
Department of Behavior and Cognitive Neuroscience
School of Psychology
Shaanxi Normal University

Received: 03-Jun-2022, Manuscript No. AACNJ-22-67413; Editor assigned: 06-Jun -2022, PreQC No. AACNJ-22-67413(PQ); Reviewed: 20-Jun-2022, QC No. AACNJ-22-67413; Revised: 24-Jun-2022, Manuscript No. AACNJ-22-67413(R); Published: 28-Jun-2022, DOI: 10.35841/aacnj-5.3.113

Citation: Setubal J. Role of combined cognitive bias hypothesis in anxiety. J Cogn Neurosci. 2022;5(3):113

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The ability to manage unwanted memories is crucial for emotional regulation and maintaining mental state. Previous evidence indicates that suppressing retrieval, which recruits executive control mechanisms to forestall unwanted memories entering consciousness, can cause forgetting, termed Suppression-Induced Forgetting (SIF). Since these executive mechanisms involve multiple mental operations, we hypothesize that the efficacy of SIF is also limited by individuals’ capacity limitation of cognitive control.


Cognitive neuroscience, Physiological psychology


Participants were assigned to 2 groups supported the median of their Cognitive Control Capacity (CCC, estimated by the backward masking majority function task) and performed the think/no-think task with electrophysiological signals recorded. The results showed that the SIF effect was observed only within the high CCC group but not within the low CCC group. In accordance, repeated suppression attempts also resulted in an exceedingly steeper reduction in intrusive thoughts within the high CCC group. Numerous studies have shown how psychosocial stress affects a person's lifetime likelihood of developing mental disorders. Furthermore, there is proof that this connection is mediated by cognitive function. A increasing body of research, however, indicates that moderate stress may have cognitive advantages that may support resilience. Hormesis is the term used to describe the idea that low to moderate levels of stress are associated with more adaptable results. The current study aims to test the hermetic relationship between low-to-moderate perceived stress and psychopathological symptoms (internalising and externalising symptoms), as well as to cross-sectionally explore the intermediary role of cognitive functioning in this effect, using a sample of young adults [1].

Furthermore, ERP analysis revealed a decrease in recollectionrelated Late Parietal Positivity (LPP) under the no-think condition within the high CCC group. A mediation analysis revealed that the reduced intrusive memories mediated the effect of CCC on SIF. These findings suggest that suppressing retrieval could reduce traces of the unwanted memories, making them less intrusive and harder to recall. More importantly, successful SIF is constrained by the capacity of cognitive control which can be wont to make sure the coordination of multiple cognitive processes during suppression [2].

Early life adversity is linked to neuropsychiatric sensitivity and an elevated risk of various mental diseases, including schizophrenia, according to a growing body of research numerous mental diseases exhibit unique symptoms that are clinically diverse and represent behavioural and cognitive dysfunctions. The ability to solve issues differently depending on the context, adapt to new situations, and find flexible ways to accomplish goals is referred to as cognitive flexibility Neuropsychiatric patients frequently have cognitive flexibility deficit. Treatment for a variety of mental diseases will advance as we better understand the neurological and molecular mechanisms that underlie cognitive rigidity. The rodent model is a practical and useful tool for conducting life-span longitudinal studies to comprehend the changes in behavioural and physiological traits brought on by early life adversity, as well as to experimentally test the causal relationship and underlying mechanisms between early life adversity and cognitive flexibility [3].

On study the potential contribution of early adversity to cognitive rigidity, researchers have employed rodents as a model system The bond between pups and their dams, which is thought to be the most crucial relationship, was a focus of several of these models.

Selective attention involves various aspects, including the method of orientation towards the placement of attention, automaticity in allocating attention, and also the capacity and talent to regulate attention Threat-related attention bias may manifest in several ways, like facilitated processing of threat, difficulty in disengaging from threat and avoidance of attention from threat, Attention bias towards threat among anxious populations may be a relatively robust phenomenon, with one meta-analysis demonstrating a combined effect [4,5].


Another meta-analytic review, specifically on eye-tracking methods, reported that anxious individuals show an increased vigilance for threat in both free viewing and visual search tasks. therein review the problem in disengaging from threat was mainly exhibited during visual search Interpretation bias refers to the tendency of resolving ambiguous information consistently during a negative or positive manner Interpretation involves various components, like interpretation generation and selection Anxiety is related to the differential generation of meaning from an ambiguous stimulus and with the preferential selection of mood-congruent meaning (Huppert Previous reviews reported that anxious adults.


  1. Roith D, Bondy C, Yakar S. The somatomedin hypothesis. Endocr Rev. 2001;22(1):53-74.
  2. Indexed at, Google Scholar, Cross Ref

  3. Crick F, Clark J. The astonishing hypothesis. J Conscious Study. 1994;1(1):10-6.
  4. Indexed at, Google Scholar, Cross Ref

  5. Friston K, Brown HR, Siemerkus. The dysconnection hypothesis. Schiz Res. 2016;176(2-3):83-94.
  6. Indexed at, Google Scholar, Cross Ref

  7. Boucher J, Osgood CE. The pollyanna hypothesis. J Verbal Learn Verbal Behav.1969;8(1):1-8.
  8. Indexed at, Google Scholar, Cross Ref

  9. Hyde JS. The gender similarities hypothesis. Am Psychol. 2005;60(6):581.
  10. Indexed at, Google Scholar, Cross Ref

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